case-study

Case Study onCerebrovascular Accident



PURBANCHAL UNIVERSITY

EVEREST COLLEGE OF NURSING













 










A
CASE STUDY
ON
Cerebrovascular Accident





Submitted By                                                         Submitted To     
Susmita K C                                                           Miss Sonam Limbu
Roll no:38                                                               Miss Alisa Risal
                                                                                Miss Anjali Shrestha



Date of submission:2073/10/




S.N       CONTENTS                                                                              PAGE NO
                                                                                          
 1          Acknowledgement
2           Background
3           Selection of Case
4           Objectives(General +Specific)
5           Bio-data of patient
6           chief complain
7           History of past illness
8            Personal history
9            Family history/Family Tree
10          Physical Examination
11          Summary of history taking & physical examination
12         Growth & Development
13          Cerebralvascular accident
14          Etiology
15           Patho-physiology
16          Clinic features
17           Diagnostic procedure
18          Management & Treatment
19          Drug Bank
20         Nursing Management
21         Application of nursing theory
22         Nursing Care Plan
23        Stress Management & Diversional Therapy
24         Discharge Planning & Teaching
26        Learning experience from case study
27        Summary
28        Bibliography










                            ACKNOWLEDGEMENT



           This case study report on Cerebralvascular accident has been prepared
based on the patient work-up during the eight weeks clinical posting at Helping hand Hospital,Chabehil, kathmandu. This report has been prepared as a partial fulfillment for the post Basic Basic Bachelor in nursing curriculum of Purbanchal University under practicum of Medical Surgical Nursing. I realized that is requirement to complete a case study has been an important opportunity for us to gain new experience and knowledge.
I could have never completed this case study without the help of some intellectual giants and wellwisher.I want to express my heartfelt gratitude and appreciation to all those people whose voluntary efforts continued to defy the received wisdom.
Specially, I would like to express all my gratitude to Co-ordinator Sonam Limbu, Instructor Anjali Shrestha and Alisha Rijal, for providing continuous monitoring, supervision, guidance and encouragement throughout the entire period of clinical practicum.
                I would like to express my gratitude to the campus chief, tutors, administration, and library staffs for providing me the opportunity to have case study and making availability of all the necessary materials. I would like to remember my seniors and colleagues for their valuable suggestions and support.
                  Here I can’t forget to express thanks to Director, Matron, all Incharge of  Helping hand Hospital for granting me the permission to conduct the case study.Furthermore I would like to thank all the concerned staffs for their help and co-operation. Without their help the case study would not have been completed.
At last but not least, my genuine gratitude is offered to the patient and her family
for providing me the valuable information, time co-operation that help me for a successful completion case study.

Susmita kc
PBBN 1st year
Everest College Of Nursing
Tinkune,Kathmandu.




Background of the study:

As a partial fulfillment of Post Basic Bachelor Nursing Curriculum Of Purbanchal University
under practicum of Medical Surgical Nursing,we are required to do 8 weeks practical at
Helping hand Hospital,Chabahil,Kathmandu.
During this practicum period,we had posting in different wards, where we individually
were supposed to do detail case study of the patient. So,I choosed case from Medical ward where
the patient was diagnosed as Cerebrovascular Accident” for the case study.

Selection of the case study on Cerebrovascular Accident:

I have chosen the case of  CVA  which is a major health problem that effect cardiovascular system showing effect in neurological disorder
So, learning about this problem deeply it’s all parts, we can educate patient’s family and
other people about this problem and its prevention.

Objective of the case study on Cerebrovascular Accident:

General Objectives
To gain depth knowledge about "CVA" by comprehensive assessment, proper nursing
diagnosis, planning appropriate care, implementing care according to need and evaluate the
outcome.

Specific Objectives
· To provide holistic nursing care to the patient by applying nursing process according to
the nursing theory.
· To apply knowledge from the basic science and nursing theories in planning the
comprehensive care to patient.
· To gain the knowledge about specific condition.
· To maintain good interpersonal relationship with the patient and family members and
provide care according to their need.
· To provide health teaching and psychological support to the patient along with the
visitors to promote and maintain the health status of the patient.
· To provide information and knowledge about the disease process to the patient and
family.
· To prevent from further complications.









Bio-Demographic Data of the Patient

Name                              Uma Devi Ghimire
Age                                 68 years
Sex                                 Female
Religion                         Hindu
Marital Status                married
Economic status            middle class family
Educational status         Illiterate
Occupation                    housewife
Ward                              Medical ward
Bed no                           05
Inpatient number          
Date of admission                 2073/9/10
Diagnosis                        Cerebrovascular Accident
Attending doctor             Dr.Niraj         
Residence                        Kapan,Kathmandu
Informants                       Patient self, her daughter and documents.

Chief complains:

Productive cough,fatigue since 3-4 days
Breathlessness since 1day
Restless, chest tightness since night

History of present illness:
This patient is the known case of copd under regular medication ,who states that she was in
her usual state of health until 3-4days prior to admission, when she experienced productive
cough with excessive mucoid to purulent sputum usually worse in morning with worsening
dyspnea doing simple activities 1day back that increases on supine position. Since night she
became restless and had chest tightness then, she was brought to Madhyapur Hospital
,Emergency department. So she was admitted in medical ward for further management.
History of past illness:                                                                                                     She doesn't have any history of surgery and other
genetic or hereditary history.

History of allergies/injuries/previous hospitalization:
No history of major injuries till now. Patient was hospitalized 1 year back due to COPD and
gives history of cold,dust allergies as characterized by cough .



Personal Habit:
Smoking: chronic smoker(for 15-20)years but now stopped since 1year.
Drinking habit: No such history.
Drugs:Under regular medication as per prescription=Tab Doxobid OD HS,R/C Foracort400mcg
BD,R/C TIOVA 18mcg OD,tab.lasilactone 1 tab P/O morning.
Dietary Habit:She had regular dietary habit and is a non-vegetarian
No.of meal taken : heavy meal morning and night ,morning breakfast and lunch at day time.
Food allergy:none
Recreation:enjoys watching TV, listening bhajans
Elimination habit:normal bowel and bladder habit
Rest and sleep habit: She used to sleep 6-8 hours at night and 1 hour day nap
Immunization: not known
Birth History:Place of delivery=Home delivery
Mode of delivery=normal
Birth weight=not known
Birth complications=not known
Bathing-1to 2 times a week
Socio economic status:
She is from middle class family.They use gas for cooking now but used to have woodfire
6-7years back.
No.of rooms in home=10
Separate kitchen room=yes
Source of drinking water=Jar water after boiling
Type of toilet=water seal latrine
Type of drainage system=closed
Type of refuse disposal=taken by municipality van
Psychological History:
She has not any psychiatric illness till now.
Health related beliefs and practices:
She has faith on modern medicines and also beliefs on traditional healer in some extent.
Occupational History:
She is a house wife takes care of her home and family.
Family History:
Type of family=joint family
They have 5 children- 2 sons and 3 daughters who all are maried. She is now living with her
husband, two sons ,daughters in law and elder son have 1 boy.Her husband have diabetes and
hypertension under medication.No known disease on her children.
Family tree:
Indicator:
EXPIRED MALE FEMALE
EXPIRED FEMALE PATIENT
MALE





PHYSICAL EXAMINATION:

A physical examination is the evaluation of a body to determine its state of health. A
complete health assessment also includes gathering information about a person’s medical history
and lifestyle, conducting laboratory tests, and screening for disease. These elements constitute
the data on which a diagnosis is made and a plan of treatment is developed.
I also performed the physical examination of Mrs.Uma Devi Ghimire to determine her health
status. The techniques used for physical examination are inspection, palpation, percussion,
auscultation, measurement and smelling.
Baseline data/measurement:
· Height: 5ft 2inches
· Weight: 62kg
· Temperature: 97.00F
· Pulse: 100/min
· Respiration: 36/min
· Blood pressure: 110/80mm of Hg
· SPO2:78% with out O2, 92%with 02
General appearance
· Consciousness level-She is conscious and oriented to time, place and person.
· Nutritional status- looks weak,thin
· Appearance- she is anxious and ill looking.
· Posture/gait-balanced
· Hygiene and grooming- poorly groomed.
· Behaviour- appropriate reaction to the situation.
· Mood-no mood swings. Appropriate to situation.
· Speech-. Language easily understandable.
· Integumentary:
· Skin colour- black brown
· Texture-dry,wrinkled skin
· Cyanosis-absent
· Turgor- intact
· Oedema- nil
· Erythema-not seen
· Hair-thining and graying of hair
· Nails-no clubbing, capillary refill normal
· Head:
· Normocephalic
· No any mass, nodules or scar
· No depressed swelling or injury.
· Scalp clear, no dandruff, lice
· Eyes:
· Eyebrows-equally distributed hair, symmetrical, equal movement of eyebrows.
· Eyelid-no exopthalmus, no ectropion and no entropion, symmetrical closing, no
swelling, redness, no drooping.
· Bulbar conjunctiva-transparent and white in colour.
· Palpebral conjunctiva-slightly pinkish in colour, no discharge.
· Sclera-white in colour.
· Cornea-transparent, no abrasions, no opacity.
· Lens-equal, light reflex is equal, clear, no opacities. Pupils-symmetrical, round
constricts on light and well accommodated. PERRLA-pupils equal, round, reacting to
light and well accommodated, prompt response to light. No sluggishness or absent of
reactivity.
· Ocular movement-smooth, symmetrical, bilateral equal movement, no divergence in
any positions/no squint present.
· Conjunctival hemorrhage-not present.
D) Ears:
· Location-normal, the margins of the pinna, outer canthus of the eye meets at occipital
protuberance.
· Pinna-no lesion, lump, smooth rounded counter and B/L symmetrical.
· External ear canal-no redness, discharge, mass or foreign body. Wax present.
· Lymph nodes and mastoid area-not palpable, no swelling and tenderness
· No discharge or drainage from the ear.Hearing normal.
E) Nose:
· Location-centrally located on face.
· Nostrils-uniform, no nasal flaring and discharge.
· Nasal septum-normal, no deviation, no extra growth.
· Nasal canal-pinkish mucosa, no extra growth.
· Smelling-good.
· Sinuses-no tenderness and pain when palpating maxillary and frontal sinuses.
F) Face:
· Symmetrical – symmetrical in terms of palpebral fissure, nasolabial folds. No facial
palsy.
· Sensation- Able to feel normally. Has touch sensation.
· Temporal pulse – Present
· Movement – Symmetrical up on voluntary movement, smile, puff out cheeks, shows
teeth.
G) Mouth and throat:
· Lips-brown in colour but dry, no injury.
· Teeth-yellowish in colour, dental caries present and no missing teeth.
· Mucosa-moist and pink in colour.
· Gums-no ulcer or inflammation. No gum bleeding.
· Tongue-cyanosed without oxygen, no ulcer, can move freely.
· Palate-soft palate(pink in colour), hard palate(white in colour)
· Tonsils-present, no swelling.
· Pharynx-pink, smooth oropharynx.
· Uvula-free hanging, single, pear shaped projection.
· Gag reflex-present.
H) Neck:
· Range of motion-normally flexed, no stiffness normal extension.
· Thyroid gland-no enlargement, no swelling, no lump on the neck.
· Jugular vein-slightly distended
· Lymph nodes-no tenderness.
· Carotid pulse-bilateral equal and strong.
I) Chest (thorax and lungs)
· Inspection
· Barrel shaped(AP diameter increased than transverse diameter) prolonged expiration
· Palpation:
- No any lump, tenderness, depression along the ribs.
· Percussion:
- High resonance on percussion
· Auscultation: expiratory wheeze, decreased breath sound,
.
J) Heart:
· Auscultation:-
- Normal lub-dub sound heard on the valves.
- Apex beat-regular in rate and rhythm, 100/min, compatible with radial pulse.
K) Back:
· Spine-centrally located, ’s’ shaped, concave on cervical region, convex on thoracic
region, concave lumbar region and convex on sacral region.
· Slightly leaned forward.
· Spinous process-vertically aligned, non-tender, muscle mass equal, no any lesions.
· Breathe sounds-wheeze present.
· Range of motion-full(with difficulty)
L) Abdomen
- No scars
- No visible blood vessels.
- Abdominal distention presentation and abdominal pain.
- Liver and spleen are not palpable.
- Gurgling bowel sound present.
M) Anus and Female genitalia
- Normal
- No abnormal discharges.
- No history of bleeding during defecation.
N) Extremities:
· Upper extremities/arms
· Skin-slightly dark
· Nails-hard, pink in colour, no clubbing of nail or thickening.
· Symmetry-symmetrical in shape and size.
· Range of motion-full in all joints, bilaterally equal.
· Joints-no tenderness, no swelling.
· Temperature- normal
· Reflexes-biceps and triceps reflex are present.
· Muscular strength present.
· Lower extremities/legs
· Skin-slightly dark, No pallor and cyanosis.
· Temperature-normal
· Nails-hard, no clubbing
· Symmetrical-symmetrical in shape and size.
· Range of motion full in all joints, bilaterally equal.
· Joints-no tenderness, no swelling.
· Pulses-dorsalispedis, posterior tibials and popliteal pulses are present.
· Reflexes-planter reflexes are present.
· Posture and gait-normal.
O) Nervous system:
- Muscle strength-equal strength in all limbs.
- Sensation-good
- Co-ordination and movement-well co-ordinated.
P) Reflexes:
· Motor and sensory reflex-good
· Corneal reflex-positive
· Biceps reflex-positive
· Triceps reflex-present
· Brachio-radial reflex-both forearm flex
· Knee-jerk reflex-present
· Planter reflex-present
· Ankle reflex-present.
Summary of History Taking and Physical Examination
History taking and physical examination helps to reveal information about the patient.
They are the tool in obtaining subjective and objective data and thus help to assemble
information about patient.
About 80% of the information in the assessment is obtained by history taking. It is
therefore essential to take history in systematic manner. About 15% of the information is
revealed by physical examination. It helps to identify the health status of the patient. Physical
examination is performed to gather objective data and to correlate them with subjective data. It
also reveals additional problems that the patient has not recognized. When doing physical
examination, cephalocaudal approach is followed, that is head to toe approach.
After performing history taking and physical examination following things were found:
· Patient was anxious, ill looking.
· Patient belonged to middle class family.
· Used to smoke but now stopped since 1 year.
· Dental carries present
· Patient is dirty and poorly groomed.
· Respiration rate 36breaths per minute
· Nutritional status-muscle wasting
· Appetite-slightly decreased.
· Dry lips, dry wrinkled skin
· Chest:expiratory wheeze, barrel shaped, hyper resonance
DEVELOPMENTAL TASKS:
My patient’s age is 68years, falls under late adulthood. WHO divides the later maturity in 3
segments:Elderly(65-75),The old(76-90),Very old>90 years.Resolving the crisis of integrity
versus despair. This is Erikson’s eighth and final developmental stage,which individuals
experience during late adulthood.In the later year of life, we look and evaluate what have done
with our lives. Through many different routes, the older person may have developed a positive
outlook in most of all of the previous stages of development.If so,the retrospective glances will
reveal a picture of a life well spent, and the person will feel a sense of satisfaction-integrity will
be achieved. If the older aduli resolved many of yhe earlier satages, negatively, the retrospective
glances likely will yield doubt or gloom- the despair Rrikson talks about.
According to the Book
In My Patient
Disengage for paid works As she was a house wife she was never
engaged in paid works.
Reassess for finances Thinks medical costs as a financial burden
Be concern with personal health care She is not maintaining maximum level of
health due weakness,doesn’t eat adeequately
Maintain interest in people outside the family
and in social ,civic and political responsibilities
She is not interested in social, civil , and
political responsibilities
Adjust to a single state Her husband is alive
Continue a supportive, warm relationship with
the spouse,including a satisfying relationship
She has good, supportive and warm
relationship with her husband
Find a satisfactory home or living arrangement
and establish a safe, comfortable household
routine to fit health.
She has good living arrangements but has
limited activity at home due to fatigue.
Be reconciled to death She has adjusted the dealth of her parents
Recognize and adjust to the aging process She accepts her ageing process and decreasing
physical strength and health.
Anatomy and Physiology of the Lung
The lungs are the primary organs of the respiratory system. The main function of the human
respiratory system is to transport oxygen from the atmosphere into the blood, and to expel carbon
dioxide from the body. Healthy levels of oxygen are absolutely crucial for the human body, as
oxygen gives our cells energy and helps them regenerate.
The Anatomy of the Lung
Each lung is divided into lobes. The right lung, which has three lobes, is slightly larger than the
left, which has two. The lungs are housed in the chest cavity, or thoracic cavity, and covered by a
protective membrane called the pleura. The diaphragm, the primary muscle involved in
respiration, separates the lungs from the abdominal cavity.
The pulmonary arteries carry de-oxygenated blood from the right ventricle of the heart to the
lungs. The pulmonary veins, on the other hand, carry oxygenated blood from the lungs to the
heart,so it can be pumped to the rest of the body.
The lungs expand upon inhalation, or inspiration, and fill with air. They then return to their
resting volume and push air out upon exhalation, or expiration. These two movements make up
the process of breathing, or respiration.
The respiratory system contains several structures. When you breathe, the lungs facilitate this
process:
· Air comes in through the mouth and/or nose, and travels down through the trachea, or
"windpipe." This air travels down the trachea into two bronchi, one leading to each lung.
The bronchi then subdivide into smaller tubes called bronchioles. The air finally fills the
alveoli, which are the small air sacs at the ends of the bronchioles.
· In the alveoli, the lungs facilitate the exchange of oxygen and carbon dioxide to and from
the blood. Adult lungs have hundreds of alveoli, which increase the lungs' surface area
and speed this process. Oxygen travels across the membranes of the alveoli and into the
blood in the tiny capillaries surrounding them.
· Oxygen molecules bind to hemoglobin in the blood and are carried throughout the body.
This oxygenated blood can then be pumped to the body by the heart.
· The blood also carries the waste product carbon dioxide back to the lungs, where it is
transferred into the alveoli in the lungs to be expelled through exhalation.
Lung Volume And Lung Capacity
Terms Symbols Description Normal
Value
Significance
Lung Volume
Tidal volume
VT or TV
The volume of air inhaled
and exhaled with each
breath.
500mLor
5≈10mL∕Kg
The tidal volume may not
vary,even with severe disease.
Inspiratory
reserve volume
IRV The maximum volume of
air that can be inhaled
after a normal inhalation.
3000mL
Expiratory
reserve volume
ERV The maximum volume of
air that can be exhaled
forcible after a normal
exhalation
1100Ml Expiratory reserve volume is
decreased with restrictive
conditions,such as
obesity,ascites,pregnancy.
Residual
volume
RV The voume of the air
remaining in the lungs
after maximum exhalation
1200ml Residual volume may be
increased with obstructive
disease
Lung capacity
Vital capacity
VC
The maximum volume of
air exhaled from the point
of maximum inspiration
VC=TV+IRV+ERV
4600ML
A decrease in vitalcpacity may
be found in neromuscular
disease,generalized
fatigue,atelectasis,pulmonary
edema,COPD & obesity.
Inspiratory
capacity
IC The maximum volume of
air inhaled after normal
expiration.
IC=TV+IRV
3500 ML A decrease in insppiratory
capacity may indicate
rstrictive disease.May also be
decreased in obesity.
Functional
residual
capacity
FRC The volume of air
remaining in th lung after
a normal expiration
FRV=ERV+RV
2300 ML Functional residual capacity
may be increased with COPD
and decreased in ARDS and
obesity.
Total lung
capacity
TLC The volume of air in the
lung after a maximum
inspiration .
TLC=TV+IRV+ERV+RV
5800ML Total ling capacity may be
decreased with restrictive
disease(atelectasis,pneumonia)
& increased in COPD
CHRONIC OBSTUCTIVE PULMONARY DISEASE
The Global Initiative for Chronic Obstructive lung Disease(GOLD)has defined chronic
obstructive pulmonary disease (COPD) as"a preventable and treatable disease with some
significant extrapulmonary effects that may contribute to the severity in individual
patients.Its pulmonary component is characterized by airflow limitation that is not fully
reversible.The airflow limitation is usually progressive and associated with an abnormal
inflammatory response of the lung to noxious particles or gases."(GOLD,2008)
COPD inlcudes emphysema, an anatomically defined condition characterised by destruction
and enlargement of the lungs alevoli; chronic bronchitis,a clinically defined condition with
chronic cough and phlegm; and small airways disease, a condition in which bronchioles are
narrowed.
COPD is present only if chronic airflow obstruction occurs.
The altenative name of COPD :
COCD- Chronic obstruction lung disease
COAD- Chronic obstruction airway disease
CORD- Chronic obstruction rspiratory disease
· ETIOLOGY
According to Book In Patient
-Cigarette Smoking Present
-Airway Responsiveness & COPD Present
-Respiration Infection Absent
-Occupation Exposures
-Air Pollution
-Antitrypsin Deficiency
-genetic
INCIDENCE AND PREVALENCE OF COPD
According to WHO estimates, 65millon people have moderate to severe COPD.At one time,it
was more common in men, but because of increased tobacco use among women in high-income
countries and the higher risk of exposure to indoor air pollution (such as biomass fuel used for
cooking and heating) in low-income countries, the disease now affects men and women almost
equally.
Total dealths from COPD are projected to increase by more than 30% in the next 10 years unless
urgent action is taken to reduce the underlying risk factors ,especially tobacco use. Estimates
show that COPD in 2030 the third leading cause of dealth worldwide.
· PATHOPHYISOLOGY
Persistent reduction in forced expiratory flow rates is the most typical finding COPD.
Increase in the residual volumme and the residual volume/total ling capacity ratio , nonuniform
distributioon of ventillation, and the ventillation-perfusion mismatching also ocurs.
- AIRFLOW OBSTRUCTION
Airflow limitation, also known as airflow obstruction, is typically determined by spirometry.
Patients with airflow obstruction related to COPD have a chronically reduced ration of FEV1
/FVC.
In contrast to asthma, the reduced FEV1 in COPD seldom shows large responses to inhaled
bronchodilators, although improvements up to 15% are common.
- HYPERINFLATION
COPD there is often "air trapping"(increased residual volume and increased ratio of residuak
volume to total lung capacity) and progressive hyperinflation(increased total lung capacity) late
in the disease.
- GAS EXCHANGE
The PaO2 usually remains normal until the FEV1 is decreased to 50% of prediction.
An elevation of the arterial level of carbon dioxide(PaCO2) is not expected until the FEV1 is
<25% of predicted.
Pulmonary hypertension severe enough to caus cor pulmonale and right ventricular failure due to
COPD typically occurs in individuals who have marked decreased in FEV1 (<25% of predicted)
and chronic hypoxemia (PaO2<55 mm of Hg).
· PATHOLOGY
Cigarette smoke exposure may affect the large airways, small airways(less or equal to 2mm
diameter), and alveoli.
Changes in large airways cause cough and sputum, while changes in small airways and alveoli
are responsible for physiologic alterations.
Emphysema and small airway pathology are both present in most persons with COPD.
- LARGE AIRWAYS
Cigarette smoking often results in mucous glaand enlargement and goblet cell hyperplasia
leading to cough and mucous production that define chronic bronchitis.
Goblet cell not only increase in number but in extent through the bronchial tree.
- SMALL AIRWAYS
The major site of increased resistance is most individuals with COPD in airways lesss than 2mm
diameter.
Characteristics cellular changes includes goblet cell metaplasia with these mucus-secreting cells
replacing surfactant-secreting Clara cells.
Infiltration of momonuclear phagocytes is also prominent.
Smooth-muscle hypertrophy may br present.
These abnormalities may cause luminal narrowing by fibrosis, excess mucus, edema, and cellular
infiltration.
Reduced surfactant may increase surface tension at the air-tissue interface,predisposing to airway
narrowing or collapse.
Because small airway patency is maintained by the surrounding lung parenchyma that provides
radial on bronchioles at point of attachment to alveolar septa, loss of bronchiolar attachments as
a result of extracellular matrix destruction may cause airway distortion and narrowing in COPD.
- LUNG PARENCHYMA
Emphysema is characterised by destruction of gas-exchanging air spaces, i.e.,the respiratory
bronchioles, alveolar ducts, and alveoli.
Their walls become perforated and latter obliterated with coalescence of small distinct air spaces
into abnormal and much larger air spaces.
· PATHOGENESIS
Airflow limitation the major physiologic change in COPD, can result from both small airway
obstruction and emphysema.
The dominant paradigm of the pathogenesis of COPD comprises four interrelated events :
(1) Chronic exposure to cigarette smoke may lead to inflammatory cell recruitment within the
terminal air paces of the lung.
(2) These inflammatory cell release elastolytic proteinases that damage the extracellular matrix
of the lung.
(3) Structural cell death resukts from oxidant stress and loss of matrix-cell attachment.
(4) Ineffective repair of elastin and other extracellular matrix components result in the air space
enlargement that defines pulmonary emphysema.
STAGES OF COPD
Stages of COPD classified on basis of spirometry result.In the spirometry lung function FEV1 is
a test that shows slow fast the client can breathe air out of the lung.FEV1 stands for forced
expiratory volume in 1 second.It can be measured by machine called spirometers.
There are various stage of COPD on the basis of spirometry result:
1.AT RISK COPD-STAGE -O:In this stage breathing test is normal but there is a possibility of
developing COPD.Mild symptoms of the condition include a chronic cough and sputum
production.
2.STAGE I MILD COPD:The breathing test shows mild airflow limitation,symptoms may
include a chronic cough and sputum production.Lungs functions FEV1 of 80% of normal or
higher.
3.MODERATE COPD STAGE 2:In this stage, the breathing test shows a worsening of airflow
limitation, usually the symptoms have increased. Lung function FEV1 of 50%to 70%.
4.SEVERE III STAGE:Lung function FEV1 30-49%.
5.STAGE IV VERY SEVERE COPD:Life threatening COPD flare ups with cyanosis,chronic
cough with lots of mucus,weight loss and severe shortness of breath.
Clinical Features:
ACCORDING TO BOOK IN MY PATIENT
Productive cough Excessive present mucoid to purulent
Shortness of breath Present when exertion also on rest
Wheezing Expiratory wheeze sound heard on auscultation
Chest tightness present
Tachypnea RR32-38breaths/min
Barrel chest AP diameter greater than transverse diameter
Clubbing of fingers of toes absent
Distended neck veins present
Fatigue Present on walking short distance,climbing
upstairs.
Hoover’s sign absent
Hypoxaemia
hypercapnia
Diagnosis of pneumonia:
ACCORDING TO BOOK IN MY PATIENT
History taking,physical examination Done
Chest X-ray done
Arterial blood gas analysis Not done
ECG done
Pulmonary function test by spirometer Not done
TIME test
Sputum G/S,C/S
Blood for renal function, CBC
Pulse oximetry monitoring
Alpha 1 antitrypsin deficiency test Not done
Laboratory finding comparison:
SN Examination Normal value Patient report
1. Hematology test:
Total leucocytes
Differential counts
Neutrophils
Lymphocytes
Eosinophils
Monocytes
Basophils
RBC counts
Haemoglobin
PCV
Mcv
MCH
MCHC
Platelets
BIOCHEMICAL TESTS
Blood glucose(R)
Urea
Creatinine
NA
K
4000-11000/mm3
40-75%
20-50%
1-6%
2-10%
0-1%
4.5-6.5million/mm3
2.
3. Neutrophils 50-70% 67
4. Lymphocytes 25-30% 27
5. Monocytes 4-6% 06
Management of copd:
· Medical management of COPD
i. Maintaining patient airway and effective gas exchange through.
a. Suctioning and liquefying secretions fluids,humidification of air.
b. Chest physiotherapy and deep breathing and coughing excersice.
c. HIgh fowler's position (promotes good lung expansion.
· Pharmacological management
i. For improve ventillation
a. Bronchodilator via inhalation,oral,intravenous.
b. Beta2 adrenergic which increased mucus clearance and dilatation of bronchus.
c. Anticholinergics-bronchodilators-beta2 against blocks the cholinergic receptors in large
airways resulting bronchodilation.(Ipravent)
d. Methylxanthines-theophylines,aminophylineshas bronchidilatory property,which
enhance mucus ciliary clearance and stimulates respiratory center.
e. Corticosteriod-decrease inflammation and edema,bronchodilatory effects.
f. Oxygen therapy-low flow/concentration of O2 1-3 lit via canula.
g. Antibiotics-if infection or severe form of COPD or an exacerbation.
· For removal of bronchial secretion
i. Nebulization with bronchodilators and positive pressure airway flow.
ii. Postural drainage and chest physiotherapy,deep breathing and coughing excersice.
· For promoting excercise
i. Aerobics excercise improves the lungs function but strengthen respiratory muscles.
ii. Breathing excercise may be prescribed,to encourage diaphragmatic breathing
discourage,rapid,shallow,panic breathing.Encourage purse lip breathing too which
enhanced CO2 flushing.
CONTROL COMPLICATIONS:
Edema and corpulmonale are treated with diuretics and digitalis.
· Improve general health/change life style.
-Stop smoking, avoid exposure to allergy and avoid high altitudes
-Adequate nutrition-frequent meals and provide more calorie diet
· Surgical management in COPD
Sometimes surgery is helpful for COPD in selected cases
-Bullectomy:surgical removal of a bulla large air filled space more in lungs
-Lung volume reduction surgery is similar, relatively good lung and work better
-Lung transplantation is sometime performed for severe COPD, particularly in
younger adults.
· Other management
-Influence vaccine:patient should be given annually to prevent influenza and
infection.
-Benzodiazepines is used in low to reduce anxiety
-In advanced critical care illness decision for CPR are addressed.
Treatment used in my patient:
· Oxygen inhalation.
· Pharmacotherapy
· Inj taxim 125 mg IV TDS
· Inj Genta 7.5 mg IV BD
· Inj Dexona 0.5mg IV TDS
· Inj Aciloc 3 mg IV TDS
· Inj 10% dextrose 180 mg over 24 hours.
On discharge:
· Syp. Sporidex 0.5ml PO TDS for 5 days.
Oxygen therapy:
Oxygen therapy – is the administration of oxygen as a therapeutic modality. It is prescribed by
the physician, who specifies the concentration, method of delivery, and liter flow per minute.
Benefits of Oxygen Therapy:
Additional Benefits of Oxygen Therapy:
· Increased clarity
· Relieves nausea
· Can prevent heart failure in people with severe lung disease
· Allows the bodies organs to carry out normal functions
Long-Term Benefits of Oxygen Therapy:
· Prolongs life by reducing heart strain
· Decreases shortness of breath
· Makes exercise more tolerable
· Results in fewer days of hospitalization
Oxygen Delivery Systems
1. Nasal Cannula
· Also called nasal prongs.
· Is the most common inexpensive device used to administer oxygen.
· It is easy to apply and does not interfere with the client’s ability to eat or talk.
· It delivers a relatively low concentration of oxygen which is 24% to 45% at flow rates of
2 to 6 liters per minute.
2. Face Mask
· It covers the client’s nose and mouth may be used for oxygen inhalation.
· Exhalation ports on the sides of the mask allow exhaled carbon dioxide to escape.
Types of Face Masks:
· Simple Face Mask - Delivers oxygen concentrations from 40% to 60% at liter
flows of 5 to 8 liters per minute, respectively.
· Partial Rebreather Mask – Delivers oxygen concentration of 60% to 90% at
liter flows of 6 to 10 liters per minute, respectively.
· Non Rebreather Mask – Delivers the highest oxygen concentration possible
95% to 100% – by means other than intubation or mechanical ventilation, at liter
flows of 10 to 15 liters per minute.
· Venturi Mask – Delivers oxygen concentrations varying from 24% to 40% or
50% at liter flows of 4 to 10 liters per minute.
3. Face Tent
· It can replace oxygen masks when masks are poorly tolerated by clients.
· It provide varying concentrations of oxygen such as 30% to 50% concentration of oxygen
at 4 to 8 liters per minute.
4 . Transtracheal Oxygen Delivery
· It may be used for oxygen-dependent clients.
· The client requires less oxygen (0.5 to 2 liters per minute) because all of the low
delivered enters the lungs.
Oxygen Therapy Safety Precautions:
· For home oxygen use or when the facility permits smoking, teach family members and
roommates to smoke only outside or in provided smoking rooms away from the client.
· Place cautionary signs reading “No Smoking: Oxygen in use” on the clients door, at the
foot or head of the bed, and on the oxygen equipment.
· Instruct the client and visitors about the hazard of smoking with oxygen use.
· Make sure that electric devices (such as razors, hearing aids, radios, televisions, and
hearing pads) are in good working order to prevent the occurrence of short-circuit sparks.
· Avoids materials that generate static electricity, such as woolen blankets and synthetic
fabrics. Cotton blankets should be used , and client and caregivers should be advised to
wear cotton fabrics.
· Avoid the use of volatile, flammable materials such as oils, greases, alcohol, ether, and
acetone(e.g. nail polish remover), near clients receiving oxygen.
· Ground electric monitoring equipment, suction machines and portable diagnostic
machines.
· Make known the location of the fire extinguishers, and make sure personnel are trained in
their use.
Drugs profile:
Cefotaxim
Action: inhibits bacterial cell wall synthesis, rendering cell wall osmotically unstable leading to
cell death.
Uses:
· Gram negative organisms- haemopilus influenza, haemophilus parainfluenzae, E.coli,
enterococcus faecals, neisseria gonorrhea, neisseria meningitis.
· Gram positive organisms- streptococcus pneumonias, streptococcus pyogenes,
staphylococcus aureus.
· Serious lower respiratory tract, urinary tract, skin, bone, gonococcal infections.
· Bacteremia, septicemia, meningitis, skin infection, CNS infection.
Dosage and route;
· Adult: IM/IV 1-2 gram 12 hourly.
· Child: 1 month-12 years. IM/IV 50 – 180 mg/kg/day in 4-6 divided doses.
· Severe infection
· Adult- IM/IV 2 gm 4 hoursly, not to exceed 12gm/day
· Child 1 month to 12 years, IM/IV 50-180mg/kg/day in 4 to 6 divided dose.
Side effect:
· CNS- headache, dizziness, weakness, paresthesia, fever, chills, seizures.
· GI- nausea, vomiting, diarrhoea, anorexia, abdominal pain, cholestasis.
· Integumentary; rash, urticaria, dermatitis.
· Respiratory- dyspnoea.
Contraindication- hypersensitivity to cephalosprins.
Nursing consideration:
· Assess the sensitivity to penicillin, other cephalosporins.
· Assess electrolytes (potassium, sodium, calcium) monthly if patient is on long term
therapy.
· Assess for bowel pattern daily, if severe diarrhoea occurs, product should be discontinue.
· Assess for anaphylaxis such as rash, urticaria, prurits, chills, fever, joint pain.
· Assess for I/V site, change cannula if swelling present.
Gentamycin:
· Functional class: anti infective
· Chemical class: aminoglycoside
Action: interfere with protin synthesis is bacterial cell by blinding to ribosomal subunit, causing
misresding of genetic load. Inaccurate peptide sequence forms in protein chain, causing bacterial
death.
Indication:
Severe systemic infection of CNS, respiratory, GI, urinary tract, bone, skin, soft tissue
caused by susceptible steains of pseudomonas aeruginosa, proteus, klebsiella, serratia, E.coli,
enetrobacter.
Contraindication: hypersensitivity to this or other amino-glycosides, fungal/viral/myocabetrial
infection.
Dosage and route:
· severe systemic infection
· Adult: I/V 3 – 6 mg /kg/day in divided doses 8 hourly.
· Child: IM/IV 2-2.5mg/kg 8 hourly
· Neonate and infant: IM/IV 2.5mg/kg 8-13 hourly.
· Neonate<1 week: I/V 2.5mg/kg 12-24 hourly.
Side effects:
· CNS: confusion, depression, numbness, tremors, seizures, muscle twisting, neurotoxicity,
dizziness, vertigo
· Cardiovascular: hypo/hypertension, palpitation, oedema
· EENT: ototoxicity, deafness, visual disturbance.
· GI: nausea, vomiting, anorexia.
· Genitor-urinary- oliguria, hematuria, renal damage, azotemia, renal failure.
Nursing consideration:
· Weight before treatment, calculation of dosage is usually based on ideal body weight but
may be calculated on actual body weight.
· Assess Intake and output, report sudden changes in urine output.
· Assess for vital signs during infusion, watch for hypoternsion, change in pulse.
· Assess IV site for thrombophlebitis including pain, redness, swelling, change side if
needed.
· Assess dehydration, high specific gravity, decrease in skin turgor, dry mucous,
membranes, and dark urine.
· Assess for overgrowth of infection including fever, malaise, redness, pain, and swelling.
Dexona:
· Functional class: corticosteroid, synthetic.
· Chemical class: glucocorticoids long acting
Action: decreased inflammation by suppression of migration of polymorphonuclear leukocytes,
fibroblasts, reversal of increased capillary permeability and lyosomal stabilization.
Uses:
· Inflammation
· Allergies
· neoplasms
· cerebral oedema
· septic shock
· collagen disorder
Dosage and routes:
· inflammation:
· Adult: PO 0.75-9mg/day in divided doses 6-12 hourly or phosphate IM
0.5-9mg/day divided 6-12 hourly or acetate IM 4-16mg 1-3 weeks.
· Child- PO 0.024-0.34mg/kg/day in divided doses, q 6-12 hourly.
· ARDS (acute respiratory distress syndrome)
· Adult IM/IV (dexomethasone sodium phosphate) 0.5-9mg/day in 2-4 divided
doses.
· Child: IM/IV (dexomethasone sodium phosphate) 0.06-0.3mg/kg/day or
1.2-10mg/m² in divided doses q6-12 hourly.
Available form:
· Dexomethasone tablets 0.25mg, 0.5mg, 0.75mg, 1mg, 1.5mg, 2mg, 4mg, 6mg
· Oral solution 0.5mg/5ml, 1mg/1ml
· Injection: acetate 8mg/ml, 16mg/ml, phosphate 4mg/ml, 10mg/ml, 20mg/ml, 24mg/ml.
Side effects:
· CNS- depression, flushing, sweating, headache, mood changes, euphoria, seizure,
insomnia.
· CV- hypertension, tachycardia, edema, cardiomyopathy.
· EENT- fungal infection, increase intraocular pressure, blurred vision, cataracts,
glaucoma.
· GI- diarrhoea, nausea, abdominal distention
Contraindication: children <2 years, psychosis, hypersensitivity to corticosteroid or
benylalcohol, idiopathic thrombocytopenia, acute glomerulonepharits, fungal infection, AIDS,
TB, glaucoma.
Nursing consideration:
Assess:
· Potassium, blood, urine glucose while long term therapy, hypokalemia and
hyperglycemia.
· Weight daily: notify prescriber of weekly gain >9 lb.
· Intake and output ration, be alert for decreasing urinary output, increasing oedema.
· Infection- fever, WBC even after withdrawal of medication.
· Mental status, affect, mood, behavioral changes, aggression.
Aciloc (ranitidine bisthmuth citrate)
· Functional class: HHistamine receptor antagonist.
Action: inhibits histamine at Hreceptor site in parietal cells, which inhibit gastric acid
secretion.
Uses:
· Duodenal ulcer.
· Gastric ulcer
· Hypersecretry condition
· Gastro esophageal reflux disease.
· Stress, ulcer, active ulcers with helicobacter pylori in combination with clari thromycin.
· Heart burn.
· Unlabeled uses: prevention of aspiration pneumonitis, upper GI bleeding, NSAID
induced ulcer prophylaxis.
Available forms: ranitidine tablet 75mg, 150mg, 300mg, solution of injection 25mg/ml,
Side effects:
· CNS- headache, sleepness, dizziness, confusion, agitation, depression, hallucination
(geriatric patient)
· CV- tachycardia, bradychardia
· GI- constipation, abdominal pain, diarrhoea, nausea, vomiting.
Contraindication: hypersensitivity.
Nursing consideration:
Assess:
· Gastric Ph (>5 should be maintained)
· Intake and output ration, BUN, creatinine.
· Mental status: confusion, dizziness, depression, anxiety, weakness, tremors, report
immediately.
· In GI, nausea, vomiting, diarrhoea.
Niko paediatric drops
· Functional class- non opoid analgesic antipyretic.
· Chemical class- non salicylate, paraaminophenol derivative.
Action: may block pain impulses peripherally that occur in responses to inhibition of
prostaglandin synthesis, does not possess anti inflammatory properties; antipyretic action results
from inhibition of prostaglandins in the CNS (hypothalamic heat- regulation center)
Uses: mild to moderate pain or fever, arthralgia, dental pain, dysmenorrhoea, headache.
Doses and routes:
· Adult and child: >12 years PO/RECTUM 325-650mg q4-6 hourly, Prn. 4g/day.
· Child 1-12 years, PO 10-15 mg/kg q4-6 hourly, maxes 5 doses/24v hours.
· Neonate: rectum 10-15mg/kg/dose q6-8 hours.
Available form: tablet, injection, syrup, suspension.
Side effects:
· CNS: stimulation, drowsiness.
· GI: nausea, vomiting, abdominal pain, GI bleeding.
· Genito urinary: renal failure, (high prolonged doses)
· Integumentry: rashes, urticaria
· Toxicity: cyanosis, anaemia, neutropenia, jaundice, seizure.
Nursing consideration:
Assess
· Hepatic studies: AST, ALT, billirubin, creatinine, prior to therapy, if long term therapy is
anticipated may cause hepatic toxicity.
· Renal studies- BUN, urine creatinine, occult blood, albumin, if patient is not long term
therapy; presence of blood or albumin indicate hepatitis.
· Intake and output ratio, decreasing output may indicate renal failure.
· For fever and pain- type of pain, location, intensity, duration.
· Provide medicine with food or milk to decreased gastric symptoms if needed.
· Shake well for suspension before a
Cefalexin (INN)
It is a first-generation cephalosporin antibiotic. It is an orally administered agent with a
similar antimicrobial spectrum to the intravenous agents cefalotin and cefazolin.
Action:
Active against strep. Pneumoniae, nesseria meningitidits, N.gonorrhoeae and
staphylococci, strep. Viridans. Less active against penicillianse producing staphylococci.
It works by interfering with the bacteria's cell wall formation, causing it to rupture, and killing
the bacteria
Dosage and route:
Oral: Treatment of susceptible infections including skin, urinary and respiratory tract
infections:
Adult: 1-2 g daily in divided doses at 6-8, 12 hour intervals, increased to 6 g in deep seated
infections.
Child: 25-100mg/kg body weight daily in divided doses. Max dose: child 4g daily
Uses: Cefalexin is used to treat a number of infections including:
· otitis media,
· streptococcal Pharyngitis,
· bone and joint infections,
· pneumonia, cellulites
· urinary tract infections
· It may be used to prevent bacterial endocarditits.
Contraindication: hypersensitivity to cephalosporines.
Special precaution: hypersensitivity to penicillins, pseudomembranous colitis, pregnancy, and
lactation.
Adverse effects
· Diarrhea, dizziness, headache.
· headache,
· indigestion,
· joint pain,
· Stomach pain (usually mild) and tiredness.
Nursing consideration:
· Assess the eyes, skin color because it can also cause yellowing of the eyes
· Assess the color of skin because it can cause red, blistered, swollen or peeling
skin
· Monitor intake and output: decreased urination;.
· Observe for anaphylaxis reaction. Symptoms of an allergic reaction include rash,
itching, swelling, or trouble breathing.
· Shack well before administering the medicine.
Nursing management:
· General management:
· Make the child lie on the side position to facilitate the drainage of the nasal
secretion.
· Keep the child comfortably warm.
· Keep him in a well ventilated room and avoid overcrowding near the child.
· Give him plenty of fluid to drink.
· Administer Oas needed. Nasal catheter and cannula is the best method to
administer O
· Specific treatment:
It consist of treating the causative organism by appropriate antibiotics, the commonly uses
antibiotics are amoxicillin, cotrimoxazole, ampicilline, penicillin.
· Symptomatic management:
· Suctioning should be done as needed to remove secretion and infected form
oropharynx and nasopharynx and to facilitate breathing.
· Steam and benzene inhalation should be given twice a day or more frequently as
needed to broken out the secretion and to facilitate coughing out.
· Paracetamole is given to fever.
· Preventive management:
· Promote breast feeding and keep warm.
· Immunize the child against infectious disease
· Keep the child away from indoor smoke and dust.
· Keep the child in well ventilated and less crowed areas.
· Encourage mother to frequent/regular antenatal check up and teach mother about
infection.
Complications of COPD
-Corpulmonale with congestive heart failure and acute respiratory failure
-Spontaneous pneumothorax due to rupture of bulla
-Peptic ulcer hyper secretion of gastric acid due to decrease 02 and increase CO2
-
Prevention
Prevention includes vaccination, environmental measures, and appropriately treating other
diseases.
Vaccination
Vaccination is effective for preventing certain bacterial and viral pneumonias in both children
and adults. Influenza vaccines are modestly effective against influenza A and B. The Center for
Disease Control and Prevention (CDC) recommends that everyone 6 months and older get yearly
vaccination. When an influenza outbreak is occurring, medications such as amantadine,
rimantadine, zanamivir, and oseltamivir can help prevent influenza.
Vaccinations against Haemophilus influenzae and Streptococcus pneumoniae in the first
year of life have greatly reduced the role these bacteria play in causing pneumonia in children.
Vaccinating children against Streptococcus pneumoniae has also led to a decreased incidence of
these infections in adults, because many adults acquire infections from children. Hib vaccine is
now widely used around the globe. A vaccine against Streptococcus pneumoniae is also available
for adults, and has been found to decrease the risk of invasive pneumococcal disease.
Environmental: Reducing indoor air pollution is recommended as is smoking cessation.
Other: Appropriately treating underlying illnesses (such as AIDS) can decrease a person's risk
of pneumonia.
There are several ways to prevent pneumonia in newborn infants. Testing pregnant
women for Group B Streptococcus and Chlamydia trachomatis, and giving antibiotic treatment,
if needed, reduces pneumonia in infants. Suctioning the mouth and throat of infants with
meconium-stained amniotic fluid decreases the rate of aspiration pneumonia.
Prognosis:
COPD usually gets gradually worse overtime and can ultimately result in dealth.It is estimated
that3%of all disability is related to COPD.The proportion of disability from COPD globally has
decraesd from 1990 to 2010 due to improved indoor air quality primarily in Asia.The overall
no.of years lived with disability from COPD,however,has increased.
The rate at which COPD worsens varies with the presence of factors thay predict a poor
outcome,including severe airflow obstruction, little ability to exercise,SOB,significantly
underweight or overweight,congestive heart failure,continued smoking,and frequent
exacerbations. Long term outcomes in COPD can be estimated using BODE index which gives a
score of zero to ten depending on FEV1, body mass index,the distance walked in six minutes,
and the modified MRC dypnea scale. Significant weight loss is a bad sign. Results of spirometry
are also a good predictor of the future progress of the disease but not as good as the BODE
index.
Nursing Interventions
o Improving airway clearance
-eliminate pulmonary irritant
-cessation of smoking
-administer bronchodilators
-mobilize patient when stable
-keep secretion liquids, liquefy
o Improving breathing patterns
-Teach about breathing resraining exercise to improve dyspnea
-Teach diaphragmatic, lower coastal, abdominal breathing
-Use of pursed lip breathing
-Keep the patient head in high position
NURSING MANAGEMENT IN MY PATIENT:
Nursing management was done by applying nursing theory of Virginia Henderson through the
nursing process which is described as follows:
NURSING THEORY APPLIED IN MY PATIENT:
The role and functions of professional nurses vary with the situation. Although there is
always a role for family and the patient, the pie wedges for team members vary in size according
to
· The problem of the patient.
· The patient’s self help ability
· The help resources.
Central to nursing that seeks to help patients toward independence is empathetic,
understanding and unlimited knowledge. The patient is an individual who requires help toward
independence. The nurse assists the individual whether ill or not, to perform activities that will
contribute to health, recovery or peaceful death, activities that the individual who had necessary
strength, will or knowledge would perform unaided. That’s why, I used this theory” Virginia
Henderson Theory”.
INTRODUCTION TO VIRGINIA HENDERSON’STHEORY:
The Henderson theory of nursing was developed by Virginia Henderson. She did not believe
that she was setting out a theory, and preferred it to be thought of as a definition. Whether it is
considered a definition or a theory, it has had a wide influence on concept and practice of
nursing.
Virginia Henderson was born on November 30, 1897, in Kansas City, Missouri. She began her
nursing education in the U.S. Army School of Nursing during World War I, from which she
graduated in 1921. She died March 19, 1996, when she was 98 years old. The definition of
nursing is the fundamental part of Henderson's theory of nursing.
Henderson defined nursing as doing things for patients that they would do for
themselves if they could, that is if they were physically able or had the required knowledge.
Nursing helps the patient become healthy or die peacefully, and also helps people work toward
independence, so that they can begin to perform the relevant activities for themselves as quickly
as possible. Rather than focus on a particular task, Henderson focused on the patient. She saw
how nursing could focus on the patient, and how it was possible to focus on developing a good
nurse-patient relationship. This deeply affected her and she believed strongly that patient-focused
nursing was the most beneficial kind of nursing for the patient.
Henderson also enumerated the 14 functions she believed to be part of basic nursing care. The
nurse should help the patient to perform the following functions
Henderson’s 14 basic needs:
CONCEPT USED BY HENDERSON:
· Human being: The patient as an individual who requires assistance to achieve health and
independence or peaceful death. The mind and body are inseparable. The patient and his
family are viewed as a unit.
· Health: The quality of health rather than life itself, that margin of mental physical vigor
that allows a person to work most effectively and to reach his highest potential of
satisfaction in life.
· Environment: She used Webster Dictionary, which defines environment as “the
aggregate of all the external conditions and influences affecting the life and development
of an organism.”
· Nursing: “The unique function of the nurse is assist the individual, sick or well, in the
performance of those activities contributing to health or its recovery (or to peaceful
death) that he would perform unaided is he had the necessary strength, will or knowledge.
And to do this, in such a way as to help him gain independence as rapidly as possible.
APPLICATION OF NURSING THEORY
NEEDS NURSING CARE
· Breathe Normally · Maintain semi fowler’s position
· Administration of Oxygen by nasal cannula.
· Check SPO2 saturation routinely.
· Nebulization given as prescribed.
· Eat, drink adequately · Encourage the patient’s mother to feed the baby
adequate breast milk.
· Elimination of bodily waste · Record of urine output and stool properly.
· Desirable movement and
position
· Explain the mother and patient party to minimize
handling the baby that need extra oxygen and
prone to infection.
· Sleep and rest. · Assess for sleep pattern.
· Change the wet napkin of the baby.
· Minimize the sound while baby is sleeping.
· Suitable Clothing · Encourage the mother to wrap the baby in clean
cloth.
·
· Maintain body temperature. · Take temperature 4 hourly to assess the patient
body temperature.
· Open the windows to maintain proper ventilation
and room temperature since it’s a hot climate.
· Keep patient clean and well
groomed.
· Encourage mother and visitors to remain clean
and tidy.
· Assist to make bedside clean.
· Frequently change the wet napkin.
· Avoid danger environment. · Maintain cross ventilation.
· Make the floor dry after cleaning by opening
window and by controlling visitors.
· Keep the oxygen cylinder in wall side and bed
locker properly in safe side.
· Keep the side rail on baby’s bed.
· Communication. · Maintain good IPR with the mother and patient
party.
· Give enough time to express feeling and listen
carefully them.
· Worship according to patient
party in their own faith.
· Baby’s mother want to worship by sacrificing
black goat in Manakamana after being recovered
from her disease but did not interrupted in it.
· Work sense
accomplishments.
My patient is neonate.
· Learn, discover and satisfy
curiosity.
· Patient’s mother was satisfied with my care and
hospital staff, so she always used to wait me to
tell her curiosity and any problem or
improvements related to her child.
NURSING PROCESS AND NURSING CARE PLANS
The nursing process is often defined as the application of critical thinking to client care activities.
The nursing process is a method of organizing and delivering nursing care. To understand its
functions, components and interactions, the nurse should have a working knowledge of the
nature of the process. A process is a series of steps or components leading to a goal, which
includes the following;
· Assessment
· Diagnosis
· Planning
· Implementation
· Evaluation
ASSESSMENT:
As for assessment I did a thorough history taking and physical assessment and also gathered
information through the current charts. Hence, I collected subjective and objective data and
made assessment of patient’s needs and problems (current and potential).
NURSING DIAGNOSIS:
· Ineffective breathing pattern related to presence of tracheo-bronchial secretions and nasal
secretions as evidence by increasing respiration rate, nasal flaring and shortness of breath.
· Impaired gas exchange r/t collection of secretions affecting oxygen exchange across
alveolar membrane as evidence by dyspnea, tachycardia
· Risk for infection (spread) related to inadequate secondary defenses(decrease
hemoglobin, hematocrit and immunosuppression
· Altered in body temperature (fever) related to related to presence of infection as evidence
by Flushed skin, skin warm to touch, with body temperature of 38.4ºC
Knowledge deficit regarding home care and preventive health measures
S.N Assessment Nursing
Diagnosis
Nursing
Goal
Plan
Action
Nursing
Intervention
Rationale Evaluation
1. Subjective
data
Patient
says,’’I have
difficulty in
breathing.’’
Objective data
Wheeze on
auscultation,
tachypneac,
cough.
Ineffective
airway
clearance
related to
excessive
secretions
as
evidenced
by
difficulty in
breathing
and
ineffective
cough
The patient
Will
maintain a
patent
airway after
nursing
intervention
In my duty
hour
Assess
patient
condition
Maintain
proper
position
Provide
02 therapy
Teach
proper use
Assessed for
breathing
pattern,rate,
abnormal
sound
,forceful
respiration.
Patient was
placed on
semi-fowlers
Position
Humidified 02
via nasal
prong @2-3
litres per min
provided
Patient was
taught for
It will help
To obtain
baseline
data for the
further
management
It facilitates
respiration
by
maximum
air exchange
and lungs
expansion
It will
correct
hypoxaemia
It improves
ventilation
Goal was
partially met
as she
demonstrates
diaphragmatic
breathing,
shows signs
of decreased
respiratory
efforts
of
incentive
spirometry
and deep
breathing
and
coughing
exercise
Provide
broncho
dilators
Encourage
for
hydration
unless
contraindi
cated.
Avoid
bronchial
irritants
maximum use
of incentive
spirometry
and deep
breathing and
coughing
exercises
A:I:NS
nebulization
1:1:2 as per
prescription
provided for
10-15 mins.
Patient was
encouraged to
drink fluids
(hot)
Patient was
suggested to
avoid
exposure to
cold,
smoke,dust,
extreme
temperatures.
by opening
airways to
facilitate
clearing the
airways of
sputum. Gas
exchange is
improved
and fatigue
is
minimized.
It decreases
airway
resistance
secondary to
broncho
constriction
It helps to
keep the
secretion
moist and
easy to
expectorateI
As it causes
bronchial
Constriction
and
increases
mucus
production

S.N Assessment Nursing
Diagnosis
Nursing
Goal
Plan
Action
Nursing
Intervention
Rational Evaluation
S.N Assessment Nursing
Diagnosis
Nursing
Goal
Plan
Action
Nursing
Intervention
Rational Evaluation
S.N Assessment Nursing
Diagnosis
Nursing
Goal
Plan
Action
Nursing
Intervention
Rational Evaluation
S.N Assessment Nursing
Diagnosis
Nursing
Goal
Plan
Action
Nursing
Intervention
Rational Evaluation
Daily progress note:
2068/0
2068/03/20
2068/03/21
Today is his third day of hospitalization. His general condition is improved the day before. His
vital signs are also established. No fever and no difficulty in breathing. Morning care given in
the morning and ordered medication also given timely. Bladder and bowel were also regular. On
morning round, patient was discharge. Discharge teaching was given.
Discharge planning and teaching:
The patient needs nursing care not only during hospitalization but also after discharge from the
hospital too. So one of the important part of nursing care is discharge teaching. I gave following
instruction or teaching while discharging patient:-
· Medication to take:
Instruct and explain the patient’s mother than the medication is very important to
continue depending on the duration that the doctor ordered for total recovery of the
patient.
· Exercise:
Instruct the mother to let her child play but it should be limited to a period of time
only to prevent the occurrence of shortness of berthing.
· Treatment:
Advice the mother to keep her baby relaxes in order to recover in his present
condition. Instruct the mother to minimize the patient from exposure to open
environment such as dusty and smoke area, which airborne microorganism are
present that can be a high risk factor that may cause severity of his condition.
· Health education:
Encourage and explain to the patient’s mother that it is important to maintain proper
hygiene to prevent further infection. Instruct the patient’s mother to bath her baby
every day and explain that bathing in the morning is not a factor cause of having
pneumonia. Instruct to increase fluid intake of the patient.
· Outpatient follow up:
Regular consolation to the physician can be factor for recovery and to asses and
monitor patient’s condition.
· Diet:
Diet as tolerated, meaning, the patient can eat everything until he can. Diet play a
big role in fast recovery so that, instruct the mother to give nutritious foods intended for
respiratory problems.
Follow-up care and home visit:
Follow-up care is important for continuity of care of patient after discharge. From follow-up
care, we came to know the prognosis of disease. The main objectives of follow-up care are:
· To assess the health status of the patient.
· To find out the prognosis of disease.
· To help the patient and family to manage their problems related to health.
· To find out if there is another health problem.
· To evaluate the knowledge, that is learning in hospital.
· To prevent from further complication.
· To provide health education for promotion of and maintenance of the health of the patient
and family.The patient has no further problem related to health. The family of the patient is
more conscious to prevent the etiological factors for COPD.
Action to minimize the stress of illness and hospitalization
Hospital is a very threatening and stressful to everybody. Being a nurse, it is our duty and
responsibility to help and support them coping with the disease condition and such terrible
situation.So I took following action to minimize stress of patient party, built a good rapport with
patient and family.
· Gave complete orientation of ward, staffs, ward routine, hospital’s rules and
regulation to the patient party.
· Gave clear information on disease process, diagnostic procedures to the
mother and patient party.
· Patient and visitors were allowed to express feelings, fears and concerns
about disease.
· One visitor was allowed to stay with her to explore problems, and maintain
homely environment.
· Each procedure performed was fully explained to her and her family.
· Taught her about relaxation technique.
· Allow to follow their traditional and cultural belief during treatment .
Learning from the case study:
Case study is the effective method of learning about the related disease in depth and practice.
Case study gives the comprehensive study of one selected patient and comparison with book in a
real situation.
During my case study of COPD, I collected information from different sources such as library,
internet and consulted with doctors, seniors of ward, teachers and with my friends. I learned
many things from the case study which are as follows:-
· About patient: During case study, I was completely involved and attached with my patient
and his family members. I came to know the emotional status and medical reaction of the
patient’s family’s members about treatment and disease process.
· About family and environment: I also got an opportunity to learn general attitude of family
and their environment. I also got a chance to know socio cultural, educational and religious
and economic status of patient.
· About nursing care: I got opportunity to learn application of nursing theory while caring the
patient and use of nursing process. It enhances the scientific method of caring the patient.
· About diversional therapy and stress management: During case study, I got chance to detect
the stressful factors and different therapy to overcome these stress. I got chance to use
diversional therapy in practical.
· About documentation: During case study I also developed further skills in documentation in
a more revised manner.
· About hospital policy: During case study, I was involved in many sectors of activities like
reporting, recording, admission, discharge procedure, investigation. So I got a lot of
knowledge about hospital policy.
Finally, I think the case study is one of the ways to develop individual
knowledge and attitude.
Conclusion:
Case study is one of the most important parts of nursing practice. It is a best method of learning.
Case study is concerned with the individualized care which helps to provide holistic nursing care
including physiological, psychological social and cultural traditional beliefs.
According to the curriculum of PBBN, I had taken a case of COPD of Uma Devi
Ghimire, for case study. I collected essential health history from patient and her family during
the case study. Then thorough physical examination was done and recorded. I revised the normal
development process of late adulthood. I reviewed the collected health history, investigations
report, and outcome of physical examination and formulated nursing diagnosis. I applied the
nursing theory of Virginia Henderson for the nursing management of the patient. Complete
nursing care was provided to the patient by applying nursing process. During the case study, I
also studied about disease, its etiology, pathophysiology, its sign and symptoms, diagnostic
procedure, therapeutic as well as nursing management from different books.
Patient was admitted on 2073/1/17 with complain of cough,shortness of breath and was
treated with I\V antibiotics, Vitals were taken regularly. Patient’s general condition improved
gradually. She was hospitalized for 4 days and was discharged on 2073/1/20.
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